Short-term intense exercise training reduces stress markers and alters the transcriptional response to exercise in skeletal muscle

Keyphrases

• Active Subject 33%
• Acute Exercise 33%
• Adaptation 33%
• Cellular Environment 33%
• Cellular Proteins 33%
• Cellular Stress 66%
• Cycling Performance 66%
• Endurance Training 33%
• Enhanced Degradation 33%
• Exercise Training 100%
• Exercise-induced 66%
• Heat Shock Protein 70 (HSP70) 33%
• Intense Exercise 100%
• Intense Training 33%
• JNK Phosphorylation 33%
• Manganese Superoxide Dismutase (MnSOD) 33%
• Molecular Factors 33%
• MRNA Expression 66%
• MuRF1 66%
• Muscle Stability 33%
• Muscle Stress 33%
• Nuclear factor-κB 33%
• PGC-1 33%
• Protein Content 66%
• Protein Oxidation 33%
• Protein Stability 66%
• Recreationally Active 33%
• Reduced Stress 100%
• Response Exercise 100%
• Signal Response 33%
• Skeletal muscle 100%
• Skeletal muscle Biopsy 33%
• Stress Markers 100%
• Stress Stability 33%
• Time Trial 33%
• Transcriptional Response 100%
• VO2max 33%

Biochemistry, Genetics and Molecular Biology

• Alpha Oxidation 25%
• C-Jun N-Terminal Kinases 25%
• Cell Stress 50%
• Endurance Training 25%
• Exercise 100%
• Heat Shock Protein 25%
• Messenger RNA 50%
• Muscle Stress 25%
• Protein Content 50%
• Protein Stability 50%
• Skeletal Muscle 100%
• Superoxide Dismutase 25%
• Tumor Necrosis Factor 25%

Neuroscience

• C-Jun N-Terminal Kinases 25%
• Cell Protein 25%
• Cell Stress 50%
• Heat Shock Protein 70 25%
• Manganese Superoxide Dismutase 25%
• Protein Stability 50%
• Skeletal Muscle 100%