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Role of TGF-β in RA-induced cleft palate in CD-1 mice
Sigmund J. Degitz
, Dale Morris
, George L. Foley
,
B. Magnus Francis
Entomology
Research output
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peer-review
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Keyphrases
Retinoic Acid
100%
Transforming Growth Factor-β
100%
Acid Activation
100%
Cleft Palate
100%
CD-1 Mice
100%
Steady State
25%
TGFBR3
18%
Retinoids
12%
Mesenchyme
12%
Gestational Day
12%
Acid Treatment
6%
Cell Proliferation
6%
Morphogenesis
6%
Protein Level
6%
MRNA Level
6%
Embryogenesis
6%
Extracellular Matrix Deposition
6%
Differentiation Matrices
6%
Experimental Manipulation
6%
Acid Exposure
6%
Palate Development
6%
Secondary Palate
6%
Palatal Shelf Elevation
6%
Differential Regulation
6%
Gravid
6%
Biochemistry, Genetics and Molecular Biology
Tretinoin
100%
Retinoic Acid
100%
Transforming Growth Factor Beta
100%
CD-1 Mouse
100%
Messenger RNA
50%
Steady State
33%
TGF Beta 1
33%
Transforming Growth Factor, Beta 3
25%
Isoform
16%
Mesenchyme
16%
TGF Beta 2
16%
Retinoid
16%
Embryogenesis
8%
Morphogenesis
8%
Mediator
8%
Cell Proliferation
8%
Secretion (Process)
8%
Pharmacology, Toxicology and Pharmaceutical Science
Retinoic Acid
100%
Transforming Growth Factor Beta
100%
CD-1 Mouse
100%
Cleft Palate
100%
Messenger RNA
50%
Transforming Growth Factor Beta1
33%
Transforming Growth Factor Beta3
25%
Retinoid
16%
Transforming Growth Factor Beta2
16%
Immunology and Microbiology
Transforming Growth Factor Beta
100%
Palate
100%
CD-1 Mouse
100%
Steady State
40%
Transforming Growth Factor Beta1
40%
Transforming Growth Factor Beta3
30%
Mesenchyme
20%
Transforming Growth Factor Beta2
20%
Extracellular Matrix
10%
Embryo Development
10%
Secretion (Process)
10%
Cell Proliferation
10%
Morphogenesis
10%
Mediator
10%
Secondary Palate
10%