Bisphenol A and phthalates modulate peritoneal macrophage function in female mice involving SYMD2-H3K36 dimethylation

Keyphrases

• Macrophage Function 100%
• Female Mice 100%
• Bisphenol A 100%
• Peritoneal Macrophages 100%
• Phthalates 100%
• Dimethylation 100%
• H3K36me3 100%
• SMYD2 50%
• Toxicants 33%
• Phagocytic Activity 33%
• Human Exposure 33%
• Gene Expression 16%
• Macrophages 16%
• Expression Level 16%
• Further Analysis 16%
• Target Gene Expression 16%
• Enzymatic Activity 16%
• Interleukin-6 16%
• Inflammatory Cytokines 16%
• Tumor Necrosis Factor-α 16%
• Inflammatory Mediators 16%
• Inflammatory Diseases 16%
• Homeostasis 16%
• Innate Immunity 16%
• Lipopolysaccharide Stimulation 16%
• Acetylation 16%
• Environmental Exposure 16%
• Scavenger Receptor 16%
• Chronic Exposure 16%
• Environmental Toxicants 16%
• Chromatin Modification 16%
• Methyltransferase 16%
• Occupational Exposure 16%
• Phthalate Exposure 16%
• Epigenetic Mechanisms 16%
• Histone-modifying Enzymes 16%
• Plastics Industry 16%
• Epigenetic Inhibitors 16%
• Phenotypic Programming 16%

Pharmacology, Toxicology and Pharmaceutical Science

• Mouse 100%
• Phthalic Acid 100%
• 4,4' Isopropylidenediphenol 100%
• Cytokine 16%
• Tumor Necrosis Factor 16%
• Inflammatory Disease 16%
• Interleukin 6 16%
• Lipopolysaccharide 16%
• Scavenger Receptor 16%
• Long Term Exposure 16%
• Histone 16%
• Methyltransferase 16%
• Environmental Exposure 16%
• Occupational Exposure 16%