At the crux of maternal immune activation: Viruses, microglia, microbes, and IL-17A

Medicine and Dentistry

• Pathogen 100%
• Microglia 100%
• Disease 100%
• Virus Activation 100%
• Infection 50%
• Sequela 50%
• Nerve Cell Differentiation 50%
• Cell Signaling Pathway 50%
• Prenatal Exposure 50%
• Influenza Virus 50%
• Virus Infection 50%
• Seasonal Influenza 50%
• Prenatal Development 50%
• Neuropathology 50%
• Analeptic 50%
• Cortical Malformation 50%
• T Helper 17 Cell 50%
• Immunostimulating Agent 50%
• Offspring 50%
• Interneuron 50%

Keyphrases

• Virus 100%
• Microbes 100%
• Microglia 100%
• Interleukin-17 (IL-17) 100%
• Maternal Immune Activation 100%
• Signaling Pathway 20%
• Inflammation 20%
• Biologically Relevant 20%
• Activation Model 20%
• Neuropathology 20%
• Fetus 20%
• Neurodevelopmental 20%
• Neurodevelopment 20%
• Interneuron 20%
• Functional Phenotype 20%
• Preclinical Animal Models 20%
• Pathogen Infection 20%
• Microglial Cells 20%
• Prenatal Exposure 20%
• Offspring Behavior 20%
• Inflammatory Pathways 20%
• Immune Signaling 20%
• Stimulants 20%
• Popular Use 20%
• Environmental Risk Factors 20%
• Fetal Brain 20%
• Endogenous Microorganisms 20%
• Neuropsychiatric Disorders 20%
• Prenatal Development 20%
• T Helper 17 (Th17) 20%
• Neurodevelopmental Disorders 20%
• Infections In Pregnancy 20%
• Maternal Viral Infection 20%
• Seasonal Influenza Viruses 20%
• Microbial Signals 20%
• Immunostimulant 20%
• Cortical Malformation 20%
• Microbial Signaling 20%

Immunology and Microbiology

• Infectious Agent 100%
• Microglia 100%
• Virus Activation 100%
• Microorganism 100%
• Viral Disease 50%
• Influenza Virus 50%
• Nerve Cell Differentiation 50%
• T Helper 17 Cell 50%
• Immunostimulant 50%
• Prenatal Development 50%
• Interneuron 50%
• Offspring 50%